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Herpes simplex virus (HSV)
gains entry through abraded
skin or mucosal tissue.
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HSV is adept at penetrating
the dermis and reaching
the nerve pathways.
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HSV travels nerve pathways
to the nerve routes (ganglia).
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An active phase of HSV
infection can produce marked
skin lesions with viral shedding.
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Viral shedding can
occur without symptoms
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Viral replication can be
blocked by antiviral drugs
that disprupt DNA.
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